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The Bone and Fat
Connection, new perspectives on understanding age-related bone loss
Current Opinion in Rheumatology (In press)
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Inhibition
of Protein Farnesylation Arrests Adipogenesis and affects PPARγ Expression
and Activation in Differentiating Mesenchymal Stem Cells
PPAR Research,
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Alendronate has an Anabolic
Effect through the Differentiation of Mesenchymal Stem Cells
Journal of Bone and Mineral Research,
2007 Oct;22(10):1603-11.
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Age-Related
Changes in Lamin A/C Expression in Cardiomyocytes
American Journal of Physiology-Heart and Circulatory Physiology,
Am J Physiol Heart Circ
Physiol. 2007 Sep;293(3):H1451-6
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As a matter of fat: New
insights on the pathophysiology of senile osteoporosis
BoneKEY Osteovision,2007 4: 129-140.
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Age-related changes in
lamin A/C expression in the osteoarticular system: laminopathies as a
potential new aging mechanism.
Mechanisms of Aging
and Development, 2006 Apr;127(4):378-83. Epub
2006 Jan 30
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Vitamin D in the aging musculoskeletal system: an authentic strength
preserving hormone
Molecular Aspects of Medicine,
Volume 26, Issue 3 , June 2005, Pages 203-219
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Molecular Aspects of Medicine,
Volume 26, Issue 3 , June 2005, Pages 181-201
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1,25(OH) 2D3
acts as a bone forming agent in the hormone independent Senescence Accelerated Mouse (SAM-P/6).
American Journal of Physiology:
Endocrinology and Metabolism. 2005 Apr;288(4):E723-30. Epub 2004 Nov 30.
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Vitamin D
treatment of senescence accelerated mice (SAM-P/6) induces several
regulators of stromal cell plasticity.
Biogerontology, 2004, 6: 421-429
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Preface: Taking musculoskeletal aging out of the bench: Do we finally
understand frailty?
Molecular Aspects of Medicine. Issue on the
Molecular Mechanisms of Aging Bone.
Volume 26, Issue 3 , June 2005, Pages 141-143
Gustavo Duque, MD, PhD-Guest Editor
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Vitamin D inhibits Fas ligand-induced apoptosis in human osteoblasts
by regulating components of both the mitochondrial and Fas-related
pathways
Bone, 2004, 35: 57-64
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1,25(OH)2D3
inhibits bone marrow adipogenesis in senescence accelerated mice (SAM-P/6)
by decreasing the expression of peroxisome proliferator-activated receptor
gamma 2 (PPAR 2)
Experimental Gerontology, 2004, 39(3): 333-338.
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Will reducing adipogenesis in bone increase bone mass?: PPARgamma2 as a key
target in the treatment of age-related bone loss.
Drug News & Perspectives. 2003
Jul-Aug;16(6):341-6.
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Estrogens (E2) regulate expression and response of
1,25-dihydroxyvitamin D3 receptors in bone cells: changes with
aging and hormone deprivation
Biochem Biophys Res Commun. 2002 Dec 6;299(3):446-54.
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Age-related bone loss: old bone, new facts.
Gerontology. 2002 Mar-Apr;48(2):62-71.
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